|
Fig 7-7A
Chronic high Pi diet |

Fig 7-7B Acute low Pi diet |

Fig 7-7C
Colchicine and high Pi diet |

Fig 7-7D Colchicine and low Pi
diet |
|

Fig 7-8A PTX effects |

Fig 7-8B
PTX & PTH effects |

Fig 7-20 Histologic appearance of
trabecular bone from patient with oncogenic osteomalacia |

Fig 7-21 Microscopic appearance of bone
section from patient with vitamin D deficiency |
|

Fig 7-32
Cross section of calcified artery |

Fig 7-35 Complications of the use of
aluminum-based phosphate binders |

Fig 7-36 Same images as Fig 7-35
but under polarizing lenses |

Fig 7-37
Positive aluminum stain of the trabecular surface of Fig
7-35 |
|

Fig 9-1 Early postinfectious
glomerulonephritis |

Fig 9-2
Large epithilial cresent |

Fig 9-3 Urine sediment of patient with
acute renal failure |

Fig 9-4
An early thrombus |
|

Fig 9-5 A parenchymal infarct |

Fig 9-6A
A fine needle aspirate in renal infarction - low
maginfication |

Fig 9-6B
Diffusely degenerated and necrotic cells |

Fig 9-7 Small artery with severe
inflammation - vessel vasculitis |
|

Fig 9-8
Small artery with endothelial activation |

Fig 9-9 A cast of necrotic tubular
cells in urine sediment |

Fig 9-10 Interstitial nephritis with
edema and mononuclear inflammatory infiltrate |

Fig 9-11 Tubulitis with infiltration of
mononuclear cells into the tubular epithelium |
|

Fig 9-12 Polymorphonuclear leukocytes
forming a cast |

Fig 9-13A Fine-needle aspirate of acute
infectious interstitial nephritis |

Fig 9-13B A neutrophil contains
phagocytosed bacteria within the cyoplasm |

Fig 9-14
Numerous PML in urine sediment - acute pyelonephritis |
|

Fig 9-15A Fine-needle aspirate -
intrarenal cytomegalovirus infection |

Fig 9-15B
Tubular epithelial cells stained with antibody |

Fig 9-16 Numerous eosinophils in an
interstitial inflammatory infiltrate |

Fig 9-17A Fine-needle aspirate of acute
allergic interstitial nephritis |
|

Fig 9-17B Higher magnification of Fig
9-17A |

Fig 9-18 Severe vacuolization of
tubular cells in injured tubular epithelium |

Fig 9-19 Necrotic tubular cells and
cell debris in tubular lumina |

Fig 9-20 Sites of cell exfoliation,
attenuation of remaining cells and reactive and regenerative changes |
|

Fig 9-21 Outer medulla shows in situ
cell necrosis and loss in medullary thick ascending limb |

Fig 9-22A
Aspirate showing acute tubular cell injury and necrosis |

Fig 9-22B Fig 9-22A x 160 |

Fig 9-23 Urine sediment from patient
with acute tubular injury |
|

Fig 9-24A Myoglobin casts in the
tubules of a patient who abused cocaine |

Fig 9-24B
Immunoperoxidase stain for myoglobin |

Fig 9-25
Apoptosis of tubular cells following tubular cell injury |

Fig 10-4 Tubules with lymphocytic
invasion |
|

Fig 10-9 Antibody-mediated rejection |

Fig 10-10 Acute tubular necrosis in the
allograft |

Fig 10-11
Tubule in center - case of acute tubular necrosis in
allograft |

Fig 10-12 Calcium oxalate crystals seen
under polarized light |
|

Fig 10-16
Bland hyaline arteriolar thickening of donor origin
|

Fig 10-18
Subclinical rejection
|

Fig 10-20B
Staining of peritubular capillaries for VCAM-1
|

Fig 11-5A
Intratubular obstruction by cell debris
|
|

Fig 11-5B
Striking regeneration process is observed
|

Fig 11-5C
In the cortical interstitial compartment
|

Fig 11-13
Intravascular coagulation in a cyclosporine-treated
renal transplant recipient
|
|